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| Unit 7: Demos |
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Schematic diagrams of transport systems Single
v. double circuit circulation Structure
of the human heart (optional) Exchange of fluids at the capillaries (9c) Bacteria linked to atherosclerosis (optional) Cornell Lake
Source Cooling Biosynthesis (14d) Consequences of alcohol consumption (14f) Nitrogenous wastes (19a) Kidney function (21a) - note, the DVD on Kidney Function is optional and may only be necessary if you are confused about the objectives. |
Complications of Excessive Alcohol Consumption Complications of excessive alcohol consumption stem largely from excess hydrogen and from acetaldehyde. Hydrogen produces fatty liver and hyperlipemia, high blood lactic acid and low blood sugar. The accumulation of fat, the effect of acetaldehyde on liver cells and other factors as yet unknown lead to alcoholic hepatitis. The next step is cirrhosis. The consequent impairment of liver function disturbs blood chemistry, notably causing a high ammonia level, which can lead to coma and death. Cirrhosis also distorts liver structure, inhibiting blood flow. High pressure in vessels supplying the liver may cause ruptured varices and accumulation of fluid in abdominal cavity. There are individual differences in response to alcohol; in particular, not all heavy drinkers develop hepatitis and cirrhosis.
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